Neurological Deficits in Patients With Celiac Disease

Abstract

Celiac disease (CD) is an autoimmune enteropathy triggered by the ingestion of gluten in genetically susceptible individuals. Developments in the understanding of the pathogenetic basis of the disease and the introduction of serological diagnostic markers have enabled the delineation of its epidemiological features and its clinical spectrum. This, in turn, has led to increased interest in the possible neurological manifestations and involvement in patients with this disorder.